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Administering 75 mg/kg/day of FA to high-fat-fed LDL receptor-deficient (LDLR/) mice for 16 weeks resulted in a significant inhibition in atherosclerosis plaque progression and vascular smooth muscle cell (VSMC) dedifferentiation by modulating the mTOR/p70S6K signaling pathway, which involved the reduction in lipid levels and the suppression of oxidative stress and inflammatory response [82]

These findings confirm that the absence of dose-response is robust to landmark conditioning

As the SnCE1 tetramer formation is structurally incompatible with SUMO1 binding at S1, we hypothesize Lys231-acetylation of SnCE1 promoting its deSUMOylase activity by stabilization of the SnCE1 monomeric state (Fig

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