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mellifera venom-induced sinusoidal and centrilobular congestion, eosinophilia, cytoplasmic vacuolation, intraparenchymal hemorrhage, centrilobular necrosis, and apoptosis ( Elevated MDA and GSH levels indicate lipid peroxidation in liver tissue, a process mediated by ROS generated through various mechanisms, including neutrophil activity ( Oxidative stress induced by bee venom contributes to acute kidney injury

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Rare but theoretical concerns include glucose intolerance in predisposed individuals and the theoretical risk of stimulating pre-existing subclinical malignancy via IGF-1 elevation, a risk shared by all GH-axis interventions