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Allendorf, Kepal Patel, Insoo Suh Surgery.2024

135 Dihydroartemisinin can significantly inhibit the viability of leukemia cells, causing cell cycle G0/G1 phase arrest and ferroptosis, and the mechanism may be related to inducing autophagy and accelerating ferritin degradation by regulating AMP-activated kinase (AMPK)/mTOR/p70S6k signaling pathway

Nikbaf-Shandiz, M., Adeli, S., Faghfouri, A

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This metabolic reprogramming not only supports cell proliferation but also elevates intracellular S-adenosylmethionine (SAM) levels, promoting epigenetic alterations and ultimately inducing the acquisition of NEPC characteristics (81, 82)