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Metformin-derived carbon dots facilitate macrophage transition to the M2 phenotype by scavenging ROS and suppressing the NLRP3 inflammasome, while simultaneously blocking the IL-6/gp130 pathway to restore fibroblast homeostasis (270)

with caveola dysfunction

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It has been shown, for example, that the activation of NF-B by a variety of agents can be blocked by NAC, suggesting that the production of ROS may act as a common pathway for a diverse range of stimuli [13, 14, 54, 55, 58, 62, 101, 103, 114116]

To view a copy of this licence, visit About this article Cite this article Teruya, M., Pblio Rabello, J., Oliveira Guardalini, L