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10.1002/0471264180.os058.08
[DOI] [PubMed] [Google Scholar] Azad N, Rojanasakul Y, Vallyathan V
The data described herein are included in published articles, or experimental data may be provided upon reasonable request
The binding of C-Met to HGF initiates the receptors innate kinase activity, followed by the formation of a multisubstrate docking site for the intracellular junction protein, which recruits signaling molecules due to homologous dimerization and autophosphorylation at Tyr1234 and Tyr1235 and subsequent phosphorylation at Tyr1349 and Tyr1356 at the c-terminal tail (Trusolino et al., 2010)
