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Acknowledgements Our research was supported by the National Institutes of Health through The University of Texas MD Anderson Cancer Centers Support Grant CA016672, National Cancer Institute grant RO1CA 089266 (MHL), Directed Medical Research Programs Department of Defense Synergistic Idea Development Award BC062166 (SCY, MHL), the Susan G

In contrast, modern humans developed unique lifestyles and technologies that enabled an unprecedented extension of lifespan (Figure 2)

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Accumulating evidence underscores the central role of mitochondria in AKI pathogenesis

Many other residues will interact with NAD(P) + to hold it in place