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Moreover, also altered intrinsic excitability, synaptic connections, and chemical microenvironment appear to be at play in the LMN dysfunctions: herein, SMN loss has been closely linked to the impairment of excitatory afferent synapses in spinal sensory-motor circuits and increased Na+ channel activity, leading to LMN hyperexcitability and altered neurotransmission [32,33,34]

Cell Physiol biochem: Int J Exp Cell physiol biochem Pharmacol

Metal Ion Balance : The dipeptide functions as a natural metal-binding compound, helping maintain healthy copper and zinc balance within normal ranges to support optimal cellular function

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Vicente-Gutierrez et al., 2019