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Outcome of patients treated for myelodysplastic syndromes without deletion 5q after failure of lenalidomide therapy
To view a copy of this licence, visit About this article Cite this article Salah, R.S., Mahmoud, A.A., El-Shiekh, R.A
Approximately 2530% of AML cases show hyperactivation due to mutations in tandem duplications within genes (FLT3ITD) or in the structural domain of tyrosine kinase (FLT3TKD) [62]
however, no clear correlation with macrophage subtypes was found
