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Interestingly, as -arrestins function as physical and biochemical scaffolds that anchor and recruit endocytic machinery as well as components of the MAPK pathway to endosomes, it is possible that -arrestins may limit ERK translocation to the nucleus, thus promoting cytoplasmic ERK signaling (Bond et al., 2019)

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Results showed that at week 24, more patients treated with SC MTX achieved ACR20 (78% versus 70%) and ACR70 (41% versus 33%) responses compared to those treated with oral MTX

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