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Within the GHSR1a-MC3R dimer, the GHSR1a inverse agonist SP-analog eliminates amplification of MC3R signaling (Rediger et al., 2011), an effect likely due to the dependence on GHSR1a constitutive activity

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Several flavonoids, such as genistein (Gen), phloretin, apigenin, and daidzein (Dai), were shown to regulate the expression of GLUT1 and GLUT4 in prostate cancer depending on the cell line phenotype

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