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Classical experimental studies on reperfusion injury indicate that inflammation and oxidative stress are key determinants of secondary brain damage (Simon et al

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doi: 10.1016/s0140-6736(76)92842-7

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In conclusion, a spectrum of interventions is being developed to modulate perioperative immunity via mitochondrial pathways

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Importantly, RSS are not observed to abstract H-atoms from other molecules

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To conclude, while cuproptosis and ferroptosis have unique molecular mechanisms and traits, they might influence each other through critical signaling pathways and interactions of protein molecules within the context of SCI

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