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Exercise It is well documented that exercise programs (either aerobic, endurance, or resistance) can provide a safe therapeutic option to patients with mitochondrial myopathy, benefiting the biochemical (increasing phosphocreatine synthesis and mitochondrial enzymes) and clinical (work capacity, fatigue, quality of life, and strength) end points [54,55,56,57,58,59,60,61].Exercise has been shown to promote mitochondrial biogenesis through activation of PGC1, AMPK, P38, MAPK, and RCG-1

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