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Impaired Mitochondrial Function of Alveolar Macrophages in Carbon Nanotube-Induced Chronic Pulmonary Granulomatous Disease

When hearts are stressed by disease, energy substrates, called purines, wash out of the cell and the total pool of cellular energy becomes severely depleted

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TMAOs production depends on FMO3, and FMO3 polymorphisms may influence T2D risk (Yamazaki and Shimizu, 2013)
