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promote myocardial hypertrophy and fibrosis through the regulating the oxidation process of pyruvate and fatty acids, affecting cardiac energy metabolism and mitochondrial function, and accelerating the development of HF [113], these results suggest that TMAO may be a potential target for the treatment of HF

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Given its essential role in the detoxification of lipid hydroperoxides, and its overexpression in most of myeloid malignancies, GPX4 inhibition has emerged as a promising therapeutic strategy to specifically trigger ferroptosis and eradicate myeloid leukemia cells

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doi: 10.1038/nature744