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As part of their metabolic reprogramming, cancer cells preferentially reduce pyruvate to L-lactate, shifting from OXPHOS to glycolysis to meet their energy and biosynthetic demands, even under aerobic conditions [1] (Fig

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This interpretation is also consistent with the clinical efficacy of liver transplantation, where replacement of the main site of TTR production reduces disease progression despite continued peripheral tissue expression of TTR

Since many anti-tumor drugs have side effects that directly or indirectly affect mitochondrial function [73], these unintended effects may even be relevant for the effectiveness of the therapy