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Change history 15 September 2023 A Correction to this paper has been published: References Vu Huu K, Zangl R, Hoffmann J, Just A, Morgner N

Instead, FIN56 treatment resulted in loss of GPX4 protein through post-translational degradation, and blocked mevalonate-derived production of lipophilic antioxidants, such as coenzyme Q 10 (Figure 1)

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Furthermore, exogenous ROS inhibits the adhesion of iPSCs and induces apoptosis and senescence [284]

Its supplementation in enteral and parenteral nutrition solutions can be used to maintain high levels of GSH and prevent oxidative stress-induced damage [88]