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identified mitophagy as an intrinsic vulnerability in CRC cells subjected to statin treatment, revealing how disruption of mitochondrial homeostasis, shaped by distinct metabolic contexts, leads to cell death and therapeutic sensitivity [103] Spatial heterogeneity across colorectal tumor sites underlies distinct metabolic phenotypes [104]

Reversing Drug Resistance of Soft Tumor-Repopulating Cells by Tumor Cell-Derived Chemotherapeutic Microparticles

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opioid) or level of severity