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& Hariz, M

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Our 3% formulation uses a phospholipid-based delivery system designed to protect the GHK-Cu complex during transit through the lipid-rich outer skin layers and release it at the dermal-epidermal junction

H., et al., Idiopathic Parkinson's Disease: A Genetic and Environmental Model, Advances in Neurology 80 (1999): 215218
During stress-induced oxidation signaling, NRF2 dissociates from Keap1, avoids degradation, moves into the nucleus to begin the transcription of downstream genes