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When glutamate release exceeds glutamate uptake, the excess glutamate activates a large number of postsynaptic NMDA-Rs, resulting in the induction of excitotoxic neuronal death by allowing excessive Ca 2+ influx through the receptor-operated cation channels [73]
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10.1007/s10863-007-9101-2 [DOI] [PubMed] [Google Scholar] Lorincz MT (2010)
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