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This inhibition can decrease warfarin clearance, leading to elevated plasma drug levels and, consequently, an increased INR in patients undergoing warfarin therapy [13], [14], [17], [25], [26]

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However, in line with our observation of elevated ser/gly synthesis in HIF2 KO NSCLC cells, HIF2 deficient renal cell carcinoma (RCC) cells showed upregulation of ser/gly synthesis pathway and presented enhanced sensitivity to the inhibition of this pathway using the phosphoglycerate dehydrogenase (PHGDH) inhibitor NCT-503 [57]

Mol Plant-Microbe Interact 16:10391046 Maughan SC, Pasternak M, Cairns N, Kiddle G, Brach T, Jarvis R, Haas F, Nieuwland J, Lim B, Muller C, Salcedo-Sora E, Kruse C, Orsel M, Hell R, Miller AJ, Bray P, Foyer CH, Murray JAH, Meyer AJ, Cobbett CS (2010) Plant homologs of the Plasmodium falciparum chloroquine-resistance transporter, PfCRT, are required for glutathione homeostasis and stress responses

Regulation of inflammation by selenium and selenoproteins: impact on eicosanoid biosynthesis