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Damage to axons is mediated by the following: (1) failure in mitochondrial energy metabolism due to inhibition of the respiratory chain by nitric oxide, which in turn causes a decrease in Na + /K + ATPase activity and alters Na + dependent glutamate transporters, (2) overexpression of glutamate receptors, (3) oligodendroglial excitotoxicity, (4) massive influx of extracellular Ca ++ , (5) activation of proteases, and (6) impaired axonal transport

Nevertheless, several cellular studies have suggested the protective roles of chemical anti-oxidants, such as GSH, as well as drug metabolizing enzymes, such as NQO1 and UDP-glucuronosyltransferases (UGTs), against AQ-induced cytotoxicity (Tafazoli and OBrien, 2009

Tesamorelin is a synthetic analog of growth hormonereleasing hormone (GHRH)

Supplementation is the fastest way to increase glutathione

These signaling pathways, enzymes, and transcription factors are essential in maintaining macrophage activation and M1 polarization by driving metabolic reprogramming (17)