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doi: 10.3390/cells9071665

Structural basis for glucocorticoid receptor recognition of both unmodified and methylated binding sites, precursors of a modern recognition element

Microglial dysfunction and defective beta-amyloid clearance pathways in aging Alzheimers disease mice

In heart failure, MCU complex dysfunction causes mitochondrial calcium overload, which inhibits ETC complex I activity and triggers mtROS bursts

Given that ovarian hormones dynamically interact with core metabolic pathways as well as with neuroendocrine, immune, microbial, and behavioral systems, a mechanistic, multi-system perspective is required to understand how nutrient metabolism is regulated across the menstrual cycle, as detailed in the following section