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Description
However, a number of different pathological conditions are also associated with dysregulated GSH synthesis or alterations in its concentration, which can have potential applications as a marker for human disease [18]
More importantly, the interaction between the mitochondrial fission protein Drp1 and A induces S-nitrosylation of Drp1, which activates its GTPase activity and leads to excessive mitochondrial fragmentation
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GSH is also a key determinant of redox signaling and protection against oxidative stress
