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A well-recognized paradox in CUR pharmacology is its considerable bioactivity despite low systemic bioavailability, a phenomenon increasingly attributed to the actions of its bioactive metabolites, particularly tetrahydrocurcumin (THC) (66)

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3), the exact pattern of change over time cannot be accurately ascertained from the literature

Curcumin induced cytochrome P450 family downregulation resulting from decreased AhR translocation into nuclei attenuated effects of BaP-induced lipid accumulation and repressed cell viability