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These exosomes upregulated Wnt3a, p-GSK-3 (Ser9), and -catenin, activated Wnt/-catenin signaling, upregulated Bcl-2 and cyclin D1, inhibited Bax expression and caspase3 activity, antagonized I/R-induced cardiomyocyte apoptosis, and increased the cell survival rates

Second, the use of cis-acting variants as instrumental variables provided a clearer insight into the potential causal relationship between gene expression and disease risk

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Mature islet cells lack the expression of YAP, while overexpression of YAP in islets is associated with proliferative and antiapoptotic effects