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Because typical early atherosclerotic lesion formation observed in hyperhomocysteinemic animals can be prevented by an antiplatelet therapy with dipyridamole, the authors concluded that platelet-mediated proliferation of smooth muscle cells is a potential cellular mechanism involved in the atherogenicity of hyperhomocysteinemia

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Furthermore, the celecoxib-loaded PEA microspheres demonstrated favorable biocompatibility and extended retention in the joint cavity and is thus advantageous for OA treatment

Across all doses, GI events were reported in 41-63% of cagrilintide groups versus 32% with placebo