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It is suggested that increased NOX activity induced by SE or trauma increases steady-state levels of H 2 O 2 directly and indirectly by utilizing NADPH needed for glutathione reduction, leading to the inhibition of glycolysis and increased cell susceptibility to oxidative stress

doi: 10.2527/1997.77suppl_221x 343 WestJ

Effect of selenium supplementation in hypothyroid subjects of an iodine and selenium deficient area: The possible danger of indiscriminate supplementation of iodine-deficient subjects with selenium

There is further experimental evidence implicating UA as a protective factor in the intraneuronal deposition of alpha-synuclein

[DOI] [PubMed] [Google Scholar] Poon J.C., Josephy P.D