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FIGURE 2 7 Molecular alteration and dysregulation pathways in EC: distinction between endometrioid EC and serous EC Endometrial endometrioid carcinomas (EECs) are marked by frequent genetic mutations and pathway dysregulations that drive their development and progression (60)
Conversely, the C@PA MN (+) group showed impaired wound healing due to aggravated oxidative stress caused by RS production, as shown in Fig
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926-32211, LI-COR)
PNPLA3 and TM6SF2 variants as risk factors of hepatocellular carcinoma across various etiologies and severity of underlying liver diseases
