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( PMID: 40123421 )
Evidence suggests that a similar pathway for the metabolism of Compound A exists in humans
GSH augmentation as a potential therapeutic strategy in CNS disease has been suggested for decades, 23 although repletion efforts have been hindered by inability to assess human CNS GSH concentrations in vivo and poor oral absorption of GSH
218 Mitochondria are pivotal in regulating signal transduction, cellular metabolism, and apoptotic signals
