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The absence of an appropriate compensation by the endogenous antioxidant defense network and increased oxidative stress leads to the activation of stress-sensitive intracellular signaling pathways and the formation of gene products that cause cellular damage and contribute to the late complications of diabetes

In summary, our results reveal significant divergences in the ruminal and fecal microbiomes of offspring from different prenatal nutritional treatments despite uniform management throughout their productive cycle

The reagents used in this study are listed in Supplementary Table 1