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Such enhanced affinity may be attributed to the presence of lipid raft which facilitates enhanced interaction of prodrug with membrane transporters/receptors thereby assisting docking of the targeted ligand into the binding domain of transporter/receptor protein

Mitochondrial dysfunction leads to a loss of energy production, fatty acid accumulation, excess reactive oxygen species(ROS) generation, membrane instability, and a shift to glucose metabolism [1,2]

There appears to be an anomalous storage of mitochondrial ATP synthase subunit c that is neither the result of mutation nor enhanced expression of the protein but rather a slower degradation of the mitochondrial ATP synthase in comparison with normal cells [116]

However, the evidence is far from conclusive: Foods like carrots, cauliflower, peanuts, and even seafood produce more TMAO than red meat

(2015) 308:L344L57