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Immunosenescence is associated with dysregulated signaling pathways (e.g., overactivation of the NF-B signaling pathway and downregulation of the melatonin signaling pathway) and abnormal immune cell responses with functional alterations and phenotypic shifts
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This unprecedented precision stems from the peptide's targeting of a unique vulnerability in senescent cell biology, where FOXO4 prevents p53 from executing its normal pro-apoptotic function
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