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In many cancers, wild-type p53 is functionally inactivated through binding of negative regulators (e.g., MDM2/MDMX) to its transactivation domain (TAD), making disruption of these interactions a key therapeutic strategy

These allogeneic cells possess unique immunoregulatory capacities that calm systemic inflammation, reset dysregulated immune pathways, and aid in the repair of vascular, muscular, and connective tissue damage

[] Chronic inflammation, which is linked to the aetiology of numerous psychological disorders, is prevented by NACs anti-inflammatory properties

Am J Physiol (1989) 256(1 Pt 2):R27680

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