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[DOI] [PubMed] [Google Scholar] Kaunitz, J

A clinically defensible pathway therefore requires standardizing pre-analytics (cycle type, progesterone route, biopsy timing/site), clarifying the clinical question (timing correction vs endotype diagnosis), and integrating receptivity readouts with complementary layers when disruption is suspected, such as immune profiling and endometrial/uterine fluid proteomics/metabolomics, so that non-receptive is interpreted within a multi-dimensional endometrial endotype framework rather than as a single timing label

Comorbidities decrease physiological reserve, increasing the risk of life-threatening conditions

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The main objective is to suppress menses, thus inducing iatrogenic menopause or pseudopregnancy, which in turn reduces pain symptoms in order to prevent or postpone surgery, which is useful for long-term management of the disease