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Mol Metab (2021) 54:101389
These include replicative exhaustion due to telomere shortening, DNA damage, oncogene activation, mitochondrial dysfunction, metabolic stress, proteotoxic stress, and exposure to certain inflammatory mediators or mitogens [49]
Methods 416 , 110379 (2025)
We then found that downregulation of CSE sensitized cells to ferroptosis (Fig
doi: 10.1016/j.isci.2019.100772 463 ChenDJinDHuangSWuJXuMLiuTet al
