glutathione s transferase cooption transferases: substrates, inihibitors and pro-drugs in cancer and neurodegenerative diseases The role of glutathione-S-transferase in
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Mechanistically, SLC25A39 deficiency induced mitochondrial dysfunction, characterized by excessive mitochondrial reactive oxygen species (ROS), reduced membrane potential, diminished adenosine triphosphate (ATP) synthesis, aberrant mitochondrial permeability transition pore (mPTP) opening, and cytochrome c release
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