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The human lung is a well-aerated site, however, in some cases, such as CF, low mucociliary clearance, and thickened mucus form localized depletion of oxygen to permit overgrowth of bacteria in the human lung (Wu et al
Sci Adv 8:eabi8716
Caruso I, Cignarelli A, Sorice GP, et al
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Taken together these results, regarding the mechanism of dialysis-related fatigue, we speculate that glycolysis and fatty acid synthesis may be enhanced, at least partly, as compensatory mechanisms for ATP production in a state where mitochondrial function is impaired, as reported elsewhere 38
