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Oxidative stresses, including 4-hydroxy-2-nonenal, nitration, and oxidation, modify posttranslational -synuclein to promote -synuclein oligomerization, which inhibits oxidative stress responses as an antioxidant

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niger up to 5.49-fold of the WT, overexpression of Glr1 increased GSH content up to 2.58-fold of the WT, and overexpression of Yap1 resulted in a GSH content up to 3.54-fold of the WT (Fig
