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Oxford University Press.

In a rodent model of amphetamine-induced neurotoxicity, NAC reversed striatal dopamine depletion and significantly reduced oxidative damage markers, such as lipid peroxidation and hydroxyl radical production [99]

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The above findings strongly indicate that oxidative stress, particularly at the level of the mitochondria, plays a central role in the neuronal death that underlies a diverse group of neurodegenerative diseases

ACLS4 is also a point of regulating ferroptosis through signaling pathways (Wu et al., 2019a): E-cadherin acts through the Merlin-Hippo-Yap pathway to control sensitivity to ferroptosis by regulating expression of ACSL4 in response to cell-cell contacts