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However, the exacerbation of age-related mitochondrial oxidative stress did not affect the decline of skeletal muscle respiratory function confirming that NFE2L2 is not exclusive but part of signaling pathways that are deranged during aging [106]

The GSSG ratio of GCI/ SER2 (26.1 0.8%), GCI/ SER3 (32.0 3.3%), and GCI/ SERs (37.0 5.6%) at 48 h were higher than that of the GCI/Vector strain (22.1 0.6%)

As such, they can influence the absorption, distribution, metabolism or excretion of chemotherapeutic agents that are responsible for CIPN formation

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Balint B, Jepchumba VK, Guant JL, Guant-Rodriguez RM (2020) Mechanisms of homocysteine-induced damage to the endothelial, medial and adventitial layers of the arterial wall