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Description
FSP1 renders cells resistant to ferroptosis independent of the GPX4-GSH pathway

Mao S, Xiang W, Huang S, Zhang A

Ajoolabady, A., Lindholm, D., Ren, J

Glucocorticoid regulation of growth hormone (GH) secretagogue-induced growth response and GH secretagogue receptor expression in the rat
However, neither molecular mechanisms responsible for the APE/Ref-1 gene expression induction upon oxidative injury nor functional data regarding the real in vivo role played by post-translational modifications in controlling APE/Ref-1 functional activities are clear.24 Understanding the possible relationships existing between APE/Ref-1 levels and oxidative-stress-based human liver pathologies is of enormous importance, not only for the comprehension of the role and mechanism that APE/Ref-1 may play in the initiation and development of these pathologies but also for developing diagnostic markers and therapeutic tools
