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The cell numbers and cell viability were protected from H 2 O 2 -induced cellular senescence by PQQ, which attenuated both H 2 O 2 -induced mitochondrial respiration decreases, as well as H 2 O 2 -induced morphological and dynamic movement changes

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doi: 10.1007/s13679019-003522 46 VermaA.AzharG.ZhangX.PatyalP.KcG.SharmaS.et al

Studies in rodents have revealed significant impairment of n-3 PUFA deficiency on learning and memory (42, 43), prompting research in humans to assess the impact of omega-3 PUFA on cognitive development and cognitive decline (see Cognitive and visual development and Alzheimer's disease)