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Description
doi:10.1111/jfbc.13507
10.1152/japplphysiol.00359.2003 J
The elevated ROS production in gastric cancer can initiate genotoxic consequences, contributing to genetic instability, DNA damage, metabolic adaptation, drug resistance and occasional cell death
We investigated its effect on ferroptosis in human coronary endothelial cells treated with TSA
Wilcoxon test Kruskal Wallis test, Mann Whitney test and Fisher exact test were done for the results
