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The pathobiology of skin aging: new insights into an old dilemma

Acknowledgments We thank Vilim Simanek, in memoriam, for his guidance on writing this paper

However, the viral infections also are related with diseases in the CNS characterized by the presence of leukocyte infiltration and inflammation, inducing a progressive damage [1], such as the progressive multifocal leukoencephalopathy with the John Cunningham virus (JC), AIDS-related dementia complex observed in HIV-1-infected patients, neurodevelopmental sequelae (mental retardation, cerebral palsy, and sensorineural hearing loss) caused by congenital cytomegalovirus (CMV) infection or cerebral mass lesions in immunocompromised adults CMV-infected, and HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP) that affects the human T-cell lymphotropic virus type 1 (HTLV-1)-infected individuals

Similarly, the epidermal growth factor receptor (EGFR)-targeted osimertinib demonstrates an ORR of up to 80% [2], while human epidermal growth factor receptor- 2 (HER- 2)-targeted trastuzumab deruxtecan yields over 50% ORR in patients of HER- 2 -amplified breast cancer with brain metastases [3]

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