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however, the maximal respiration measured following the injection of a mitochondrial uncoupling agent (carbonyl cyanide-4-(trifluoromethoxy) phenylhydrazone (FCCP)) decreased significantly in the H 2 O 2 -exposed group compared to the control group, while it was protected significantly in both the 0.1 nM and 1.0 nM PQQ-pretreated groups compared to the H 2 O 2 -exposed group (Fig

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