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Because IGF-1 LR3 resists binding-protein capture, it delivers more sustained and reproducible IGF-1 receptor activation at lower concentrations, supporting cell growth, survival, and productivity

J King Saud University-Science 32:25342539 [Google Scholar] Wang Z, Sun R, Wang G, Chen Z, Li Y, Zhao Y, Liu D, Zhao H, Zhang F, Yao J (2020c) SIRT3-mediated deacetylation of PRDX3 alleviates mitochondrial oxidative damage and apoptosis induced by intestinal ischemia/reperfusion injury